Targeting miR‐155 restores abnormal microglia and attenuates disease in SOD1 mice
Abstract
To investigate miR‐155 in the SOD1 mouse model and human sporadic and familial amyotrophic lateral sclerosis (ALS).
Document Details
- Document Type
- Pub Defense Publication
- Publication Date
- Nov 27, 2014
- Source ID
- 10.1002/ana.24304
Entities
People
- Anna M. Krichevsky
- Camille E. Doykan
- David J. Greco
- Gopal Murugaiyan
- Hans Lassmann
- Howard L. Weiner
- James Berry
- Mark P. Jedrychowski
- Matthew P. Frosch
- Merit E. Cudkowicz
- Nathalie Pochet
- Oleg Butovsky
- Olga Kiner
- Pauline M. Wu
- Rachid El Fatimy
- Robert Lawson
- Ron Cialic
- Steven P. Gygi
- Susanne Krasemann
- Zain Fanek
Organizations
- ALS Association
- Edward N. and Della L. Thome Memorial Foundation
- Harvard Medical School
- Harvard University
- Medical University of Vienna
- National Institutes of Health
- National Multiple Sclerosis Society
- Race to Erase MS
- United States Department of Defense
- University Medical Center Hamburg-Eppendorf