Hepatic Deficiency of Augmenter of Liver Regeneration Predisposes to Nonalcoholic Steatohepatitis and Fibrosis

Abstract

The augmenter of liver regeneration (ALR) protein is critical for lipid homeostasis and mitochondrial function. We investigated high‐fat/high‐carbohydrate (HF/HC) diet–induced nonalcoholic fatty liver disease (NAFLD) in wild‐type (WT), hepatocyte‐specific ALR‐knockout (ALR‐H‐KO), and ALR‐heterozygous (ALR‐H‐HET) mice. ALR was measured in serum of human nonalcoholic steatohepatitis (NASH) and NASH‐induced cirrhosis (serum and liver).

Document Details

Document Type
Pub Defense Publication
Publication Date
Oct 22, 2020
Source ID
10.1002/hep.31167

Entities

People

  • Akanksha Sharma
  • Alok K. Verma
  • Chandrashekhar R Gandhi
  • Jaideep Behari
  • Jiang Wang
  • Richa Rani
  • Rohit Kohli
  • Rosa Salazar Gonzalez
  • Shimul A. Shah
  • Sudhir Kumar

Organizations

  • Children's Hospital Los Angeles
  • Cincinnati Children's Hospital Medical Center
  • Cincinnati VA Medical Center
  • University of Cincinnati
  • University of Pittsburgh

Tags

Fields of Study

  • Medicine

Readers

  • Molecular and Cellular Biology