Graphene-based sensing of oxygen transport through pulmonary membranes

Abstract

Lipid-protein complexes are the basis of pulmonary surfactants covering the respiratory surface and mediating gas exchange in lungs. Cardiolipin is a mitochondrial lipid overexpressed in mammalian lungs infected by bacterial pneumonia. In addition, increased oxygen supply (hyperoxia) is a pathological factor also critical in bacterial pneumonia. In this paper we fabricate a micrometer-size graphene-based sensor to measure oxygen permeation through pulmonary membranes. Combining oxygen sensing, X-ray scattering, and Atomic Force Microscopy, we show that mammalian pulmonary membranes suffer a structural transformation induced by cardiolipin. We observe that cardiolipin promotes the formation of periodic protein–free inter–membrane contacts with rhombohedral symmetry. Membrane contacts, or stalks, promote a significant increase in oxygen gas permeation which may bear significance for alveoli gas exchange imbalance in pneumonia.

Document Details

Document Type
Pub Defense Publication
Publication Date
Feb 27, 2020
Source ID
10.1038/s41467-020-14825-9

Entities

People

  • Cecilia Leal
  • Marilyn Porras-Gomez
  • Mijung Kim

Organizations

  • Office of Naval Research

Tags

Readers

  • Electrochemical Engineering/ Fuel Cell Technologies
  • Immunology and Pathology
  • Molecular and Cellular Biochemistry

Technology Areas

  • Microelectronics
  • Microelectronics - Graphene