Immune-checkpoint proteins VISTA and PD-1 nonredundantly regulate murine T-cell responses

Abstract

Multiple immune-checkpoint proteins, such as programmed death 1 (PD-1), LAG3, and TIM3, are coexpressed on immune cells and functionally synergize with each other. V-domain immunoglobulin suppressor of T-cell activation (VISTA) is a recently identified immune-checkpoint molecule that suppresses T-cell activation. This study establishes that VISTA and PD-1 exert nonredundant immune regulatory functions and synergistically regulate T-cell responses. Combinatorial treatment using VISTA- and PD-ligand 1-specific monoclonal antibodies achieved synergistic therapeutic efficacy in murine tumor models. This study critically advances our knowledge of the immune regulatory function of VISTA and provides a rationale for targeting both VISTA and PD-1 to more effectively treat T-cell-regulated diseases such as cancer.

Document Details

Document Type
Pub Defense Publication
Publication Date
May 11, 2015
Source ID
10.1073/pnas.1420370112

Entities

People

  • Arief A. Suriawinata
  • Austin D. Schenk
  • Halli E. Miller
  • Indira Guleria
  • Juan Putra
  • Jun Liu
  • Li Wang
  • Richard J. Barth
  • Wenna Chen
  • Yina H Huang
  • Ying Yuan

Organizations

  • China Pharmaceutical University
  • Geisel School of Medicine
  • Harvard Medical School
  • Medical College of Wisconsin
  • National Cancer Institute
  • United States Department of Defense

Tags

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Immunology
  • Oncology