Strand-biased cytosine deamination at the replication fork causes cytosine to thymine mutations in Escherichia coli

Abstract

C:G to T:A mutations constitute the largest class of spontaneous base substitutions in all organisms. These mutations are thought to be a result of cytosine deaminations, but what promotes these deaminations is unclear. We confirm here the hypothesis that they occur predominantly in single-stranded DNA (ssDNA) and identify the ssDNA in the lagging strand template as the preferred site of C:G to T:A mutations. As a consequence, replication creates a strand bias in these mutations, and this overwhelms any strand bias resulting from transcription. These results explain a long-recognized bias in base composition of microbial genomes called GC skew and predicts that C:G to T:A mutations created by the APOBEC3 family deaminases in cancer genomes should occur with the same strand bias.

Document Details

Document Type
Pub Defense Publication
Publication Date
Feb 02, 2016
Source ID
10.1073/pnas.1522325113

Entities

People

  • Ashok S. Bhagwat
  • Haixu Tang
  • Heewook Lee
  • Jesse P. Townes
  • Patricia L. Foster
  • Weilong Hao

Organizations

  • Indiana University
  • National Institutes of Health
  • United States Army
  • Wayne State University

Tags

Fields of Study

  • Biology

Readers

  • Molecular Genetics
  • Molecular and genetic basis of cancer.
  • Systems Analysis and Design

Technology Areas

  • Biotechnology