Deficits in the activity of presynaptic γ-aminobutyric acid type B receptors contribute to altered neuronal excitability in fragile X syndrome

Document Details

Document Type
Pub Defense Publication
Publication Date
Apr 01, 2017
Source ID
10.1074/jbc.m116.772541

Entities

People

  • Jayashree Chadchankar
  • Ji-yong Kang
  • John Dunlop
  • Menelas N. Pangalos
  • Michelle I. Mighdoll
  • Nicholas J. Brandon
  • Robert J. Mather
  • Stephen J. Moss
  • Tarek Z. Deeb
  • Thomas M. Hyde
  • Thuy N. Vien

Organizations

  • National Institute of Neurological Disorders and Stroke
  • United States Department of Defense