Sleep and diurnal rest-activity rhythm disturbances in a mouse model of Alzheimer’s disease

Abstract

Accumulating evidence suggests a strong association between sleep, amyloid-beta (Aβ) deposition, and Alzheimer’s disease (AD). We sought to determine if (1) deficits in rest-activity rhythms and sleep are significant phenotypes in J20 AD mice, (2) metabotropic glutamate receptor 5 inhibitors (mGluR5) could rescue deficits in rest-activity rhythms and sleep, and (3) Aβ levels are responsive to treatment with mGluR5 inhibitors.

Document Details

Document Type
Pub Defense Publication
Publication Date
May 05, 2020
Source ID
10.1093/sleep/zsaa087

Entities

People

  • Cara J Westmark
  • Carissa L Verkuilen
  • Dylan J Douglas
  • Eli Wallace
  • Lauren I Steinberg
  • Mikolaj J. Filon
  • Pamela R Westmark
  • Rama Maganti
  • Samantha Wright

Organizations

  • Fraxa Research Foundation
  • National Center for Advancing Translational Sciences
  • National Institute on Aging
  • National Institutes of Health
  • United States Department of Defense
  • University of Wisconsin–Madison

Tags

Fields of Study

  • Biology
  • Medicine

Readers

  • Circadian Sleep-Wake Regulation and Chronobiology
  • Neuroscience
  • Traumatic Brain Injury (TBI) and Cognitive Aging in the Guam and Border Populations Affected by Alzheimer's Disease and Tau-Associated Dementias.