Metabolomics Analysis Revealed Distinct Cyclic Changes of Metabolites Altered by Chronic Ethanol‐Plus‐Binge and Shp Deficiency
Abstract
Chronic ethanol (EtOH) consumption causes alcoholic liver disease (ALD), and disruption of the circadian system facilitates the development of ALD. Small heterodimer partner (SHP) is a nuclear receptor and critical regulator of hepatic lipid metabolism. This study aimed at depicting circadian metabolomes altered by chronic EtOH‐plus‐binge and Shp deficiency using high‐throughput metabolomics.
Document Details
- Document Type
- Pub Defense Publication
- Publication Date
- Oct 28, 2016
- Source ID
- 10.1111/acer.13257
Entities
People
- Li Wang
- Melanie Tran
- Suthat Liangpunsakul
- Zhihong Yang
Organizations
- Indiana University
- National Institutes of Health
- National Natural Science Foundation of China
- United States Department of Defense
- University of Connecticut
- Wenzhou Medical University
- Yale University