SARS-CoV-2 Causes Lung Infection without Severe Disease in Human ACE2 Knock-In Mice

Abstract

Mouse models of SARS-CoV-2 pathogenesis have facilitated the rapid evaluation of countermeasures. While the first generation of models developed pneumonia and severe disease after SARS-CoV-2 infection, they relied on ectopic expression of supraphysiological levels of human ACE2 (hACE2).

Document Details

Document Type
Pub Defense Publication
Publication Date
Jan 12, 2022
Source ID
10.1128/jvi.01511-21

Entities

People

  • Adolfo García-Sastre
  • Emma S. Winkler
  • Fahmida Alam
  • James Brett Case
  • Melissa B. Uccellini
  • Michael J Holtzman
  • Michael S. Diamond
  • Michael Schotsaert
  • Rita E. Chen
  • Soner Yildiz

Organizations

  • Icahn School of Medicine at Mount Sinai
  • National Institutes of Health
  • Washington University School of Medicine
  • Washington University in St. Louis

Tags

Fields of Study

  • Biology
  • Chemistry

Readers

  • Computational Modeling and Simulation
  • Infectious Disease/Epidemiology
  • Molecular Biology and Genetics