Progesterone promotes immunomodulation and tumor development in the murine mammary gland

Abstract

Clinical studies have linked usage of progestins (synthetic progesterone [P4]) to breast cancer risk. However, little is understood regarding the role of native P4, signaling through the progesterone receptor (PR), in breast tumor formation. Recently, we reported a link between PR and immune signaling pathways, showing that P4/PR can repress type I interferon signaling pathways. Given these findings, we sought to investigate whether P4/PR drive immunomodulation in the mammary gland and promote tumor formation.

Document Details

Document Type
Pub Defense Publication
Publication Date
May 01, 2021
Source ID
10.1136/jitc-2020-001710

Entities

People

  • Alfredo A Molinolo
  • Carol A Lange
  • Christy R Hagan
  • Dominika E. Helm
  • Gangjun Lei
  • Gloria M. Trinca
  • Howard H Yang
  • Junping Wei
  • Justin M. Balko
  • Katelin A. Gibson
  • Katherine R Walter
  • Kent W Hunter
  • Lauryn R. Werner
  • Margaret L Axelrod
  • Mary A Markiewicz
  • Merit L. Goodman
  • Prabhakar Chalise
  • Rashna Madan
  • Richard C Hastings
  • Sean M. Holloran
  • Xiao-yi Yang
  • Ying Hu
  • Zachary C Hartman

Organizations

  • National Cancer Institute
  • National Institutes of Health
  • Susan G. Komen for the Cure
  • United States Department of Defense
  • V Foundation for Cancer Research

Tags

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Oncology