A spatially restricted fibrotic niche in pulmonary fibrosis is sustained by M-CSF/M-CSFR signalling in monocyte-derived alveolar macrophages
Abstract
Ontologically distinct populations of macrophages differentially contribute to organ fibrosis through unknown mechanisms.
Document Details
- Document Type
- Pub Defense Publication
- Publication Date
- Oct 10, 2019
- Source ID
- 10.1183/13993003.00646-2019
Entities
People
- Alexander V. Misharin
- Alexandra C McQuattie-Pimentel
- Annette S. Flozak
- Cara J Gottardi
- Carla M. Cuda
- Ching-i Chen
- David Kirchenbuechler
- David W. Kamp
- G.r. Scott Budinger
- Harris Perlman
- Lango Sichizya
- Manu Jain
- Nikita Joshi
- Nikolay S Markov
- Paul A Reyfman
- Paul Cheresh
- Raul Piseaux-aillon
- Renea P. Jablonski
- Rohan Verma
- Satoshi Watanabe
- Ziyan Lu
Organizations
- National Heart, Lung, and Blood Institute
- National Institute of Allergy and Infectious Diseases
- National Institute on Aging
- United States Department of Defense