Neuropilin-1 is over-expressed in claudin-low breast cancer and promotes tumor progression through acquisition of stem cell characteristics and RAS/MAPK pathway activation

Abstract

Triple-negative breast cancers (TNBC) have a relatively poor prognosis and responses to targeted therapies. Between 25 and 39% of TNBCs are claudin-low, a poorly differentiated subtype enriched for mesenchymal, stem cell and mitogen-activated signaling pathways. We investigated the role of the cell-surface co-receptor NRP1 in the biology of claudin-low TNBC.

Document Details

Document Type
Pub Defense Publication
Publication Date
Jan 25, 2022
Source ID
10.1186/s13058-022-01501-7

Entities

People

  • Anja Rockstroh
  • Brett G. Hollier
  • Colleen Nelson
  • Erik W. Thompson
  • Kamil A. Sokolowski
  • Layla-rose Lynam
  • Marianna Volpert
  • Melanie Lehman
  • Philip A. Gregory
  • Yu Hin Tang

Organizations

  • United States Department of Defense

Tags

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Oncology (Cancer Research).

Technology Areas

  • Biotechnology