Mitochondrial dysfunction and consequences in calpain-3-deficient muscle

Abstract

Nonsense or loss-of-function mutations in the non-lysosomal cysteine protease calpain-3 result in limb-girdle muscular dystrophy type 2A (LGMD2A). While calpain-3 is implicated in muscle cell differentiation, sarcomere formation, and muscle cytoskeletal remodeling, the physiological basis for LGMD2A has remained elusive.

Document Details

Document Type
Pub Defense Publication
Publication Date
Dec 01, 2020
Source ID
10.1186/s13395-020-00254-1

Entities

People

  • Aditi Phadke
  • Aurelia Defour
  • Helen K. Johnston
  • Jack H. Van Der Meulen
  • Jennifer M. Peterson
  • Jessica Boehler
  • Jyoti K. Jaiswal
  • Kanneboyina Nagaraju
  • Kitipong Uaesoontrachoon
  • Qing Yu
  • Vanessa E. Jahnke

Organizations

  • Foundation for the National Institutes of Health
  • Muscular Dystrophy Association
  • United States Department of Defense

Tags

Fields of Study

  • Biology

Readers

  • Cellular and Molecular Pathways of Apoptosis.
  • Immunology and Pathology
  • Molecular Biology and Genetics