CALCIPHYLAXIS IN WOUND HEALING

Abstract

In the exploration of different calciphylactic syndromes, it was shown that a variety of agents such as distilled water, mechanical trauma, formaldehyde, croton oil, or histamine liberators selectivity inhibit skin calcification at the point where they are applied within the challenged area. We also found that in rats, topical direct calcinosis, normally induced in the subcutaneous connective tissue at the site of KMnO4-injection, is prevented if the animals are maintained on a diet virtually deficient in both calcium and phosphate. A calciphylatic syndrome characterized by heavy calcium deposits in the snout, paws and esophagus induced by certain agents was investigated. Moreover, a technique was developed for the study of mast cells. Selective necrosis was calcification at the corticomedullary junction of the kidney in the rat was produced in experiments with hexadimethrine bromide, an agent in clinical use for the inactivation of heparin.

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Document Details

Document Type
Technical Report
Publication Date
Aug 31, 1963
Accession Number
AD0408202

Entities

People

  • Hans Selye

Organizations

  • Université de Montréal

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Abstracts
  • Animals
  • Arteries
  • Biomedical Research
  • Cardiovascular System
  • Cells
  • Connective Tissue
  • Contracts
  • Dispersions
  • Fungi
  • Government Procurement
  • Mast Cells
  • Metabolic Diseases
  • Plant Oils
  • Polymers
  • Tissues
  • Wound Healing

Fields of Study

  • Medicine

Readers

  • Allergy and Immunology.
  • Immunology and Pathology
  • Toxicology/Environmental Toxicology