THE EFFECT OF SULFHYDRYL REAGENTS ON THE BINDING OF HUMAN HEMOGLOBIN TO HAPTOGLOBIN.

Abstract

Human hemoglobin was treated with the following sulfhydryl reagents: iodoacetamine, p-mercuribenzoate, cystine, cystamine, N-ethylmaleimide and bis(N-maleimidomethyl) ether (BME). Only BME hemoglobin showed impaired binding to serum haptoglobin. An excess of free BME hemoglobin was required to saturate the haptoglobin of normal serum. Competition studies indicated that BME hemoglobin bound haptoglobin about one-fourth as readily as normal hemoglobin. This finding was independent of haptoglobin phenotype. BME hemoglobin could be readily displaced from haptoglobin by an excess of 'normal' hemoglobin. Impaired binding to haptoglobin is yet another property which BME hemoglobin shares with deoxyhemoglobin. Possible mechanisms for this phenomenon are discussed. (Author)

Document Details

Document Type
Technical Report
Publication Date
Jun 12, 1967
Accession Number
AD0658968

Entities

People

  • Howard F. Bunn

Organizations

  • United States Army Medical Research Laboratory

Tags

DTIC Thesaurus Topics

  • Competition
  • Genetic Phenomena
  • Genetic Structures
  • Genetics
  • Hemoglobin
  • Phenotypes

Fields of Study

  • Chemistry

Readers

  • Data Mining and Knowledge Discovery.
  • Molecular and Cellular Biochemistry