PULMONARY HYPERTENSION RESULTING FROM OXYGEN EXPOSURE

Abstract

Rats exposed to an atmosphere of 95 to 100% oxygen at a partial pressure of 516 mm Hg for periods of 30 days showed no obvious abnormalities. At the end of this exposure they were removed from the system and anaesthetized. Aortic and intratracheal pressures were measured by standard direct procedures in response to breathing air and oxygen and to transient increases in intratracheal pressure. Compared with the results in unexposed rats, the experimental animals showed an increase in pulmonary arterial pressure (PAP) and a decrease in mean aortic pressure. The increase in aortic pressure caused by breathing oxygen which was present in the unexposed animals could be elicited in the exposed rats. The increase in PAP is attributed to structural changes in the pulmonary vasculature associated with oxygen exposure and it is postulated that an increase in vascular resistance is responsible for a decreased cardiac output, giving rise to the systemic hypotension. Whereas mean aortic pressure fell during increased intratracheal pressure, mean pressure from the right heart did not change, suggesting that the right ventricle empties itself less well against the increased pressure.

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Document Details

Document Type
Technical Report
Publication Date
Jun 05, 1968
Accession Number
AD0672755

Entities

People

  • George H. Kydd

Organizations

  • Naval Air Warfare Center Warminster

Tags

DTIC Thesaurus Topics

  • Arteries
  • Blood
  • Cardiovascular Diseases
  • Cardiovascular Physiological Phenomena
  • Cardiovascular System
  • Health Services
  • Heart
  • Hypertension
  • Laboratory Animals
  • Partial Pressure
  • Pressure Measurement
  • Pulmonary Hypertension
  • Respiration
  • Veins
  • Ventricles

Fields of Study

  • Medicine

Readers

  • Cardiovascular Physiology
  • Toxicology/Environmental Toxicology