MECHANISM OF ACTION OF BOTULINAL TOXIN ON RESPIRATION

Abstract

Botulinal toxin causes (in animals) respiratory disorders, which, at first, manifest themselves by the increased respiratory frequency and by depression of respiration, later by a decreased frequency and by a decline in the amplitude of respiratory movements, particularly in a protraction of the exhalation action. Subsequently, a recurrent respiration ensues and the paralysis of the respiratory center follows. As a result of administered botulinal toxin, changes in the reflex reaction of respiration affect the stimuli of the vagus nerve and, to some extent, those of the sciatic nerve, which is reflected in the excitability of the respiratory center during botulinal intoxication. Directly after administration of the toxin one observes an increased excitability in the respiratory centers. But, some time later following administration of the toxin, a decline in the threshold value of the respiratory center's excitability changes to an increase. Then, one can observe parabiotic phase phenomena in the respiratory center. The excitability of the phrenic nerve changes slightly during botulinal intoxication. The disorders in respiratory movements of animals during botulinal intoxication are combined with a development of the parabiotic condition in the respiratory center.

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Document Details

Document Type
Technical Report
Publication Date
Feb 01, 1966
Accession Number
AD0675832

Entities

People

  • V. N. Abrosimov

Organizations

  • United States Army Biological Warfare Laboratories

Tags

DTIC Thesaurus Topics

  • Amplitude
  • Anesthesia
  • Botulism
  • Brain
  • Diseases And Disorders
  • Frequency
  • Intoxication
  • Irritation
  • Nerves
  • Nervous System
  • Peripheral Nervous System
  • Phrenic Nerves
  • Respiration
  • Sciatic Nerve
  • Skeletal Muscle
  • Tissues
  • Vagus Nerve

Readers

  • Cardiovascular Physiology
  • Neuroscience