THE EFFECT OF PLAGUE MICROBE TOXIN ON THE ACTIVITY OF PYRUVATE-OXYDASE AND LACTATEDEHYDROGENASE IN PLAGUE-SUSCEPTIBLE ANIMALS

Abstract

Calcium pantothenate, administered subcutaneously and added in tests in vitro, intensifies oxygen consumption by liver homogenates of healthy white mice and reduces consumption of it by liver homogenates of white mice which are contaminated with plague toxin. Thiamine, administered subcutaneously and added in samples, stimulates oxidation of pyruvate by liver homogenates of healthy animals and restores to normal the oxygen consumption of liver homogenates of animals which are contaminated with the toxin of the plague microbe. Plague microbe toxin, added directly to extracts of liver, brain, and serum, does not exert an influence on lactatedehydrogenase activity. Under the influence of plague microbe toxin, administered to animals intraperitoneally, lactatedehydrogenase activity is intensified in blood serum, is suppressed in the brain, and remains without change in the liver.

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Document Details

Document Type
Technical Report
Publication Date
Jan 01, 1967
Accession Number
AD0683039

Entities

People

  • Z. I. Vasilyeva

Organizations

  • United States Army Biological Warfare Laboratories

Tags

Communities of Interest

  • Materials and Manufacturing Processes

DTIC Thesaurus Topics

  • Acids
  • Animals
  • Blood
  • Blood Serum
  • Chemical Synthesis
  • Chemistry
  • Hemorrhagic Shock
  • Intoxication
  • Lactic Acid
  • Liver
  • Oxidation
  • Pyruvates
  • Pyruvic Acid
  • Resistance
  • Rodents
  • Russian Language
  • Thiamine Pyrophosphate

Fields of Study

  • Biology

Readers

  • Cardiovascular Physiology
  • Microbial Pathology
  • Toxicology/Environmental Toxicology