ACTIVATION ENERGIES OF ACCELERATION AND HYPOXIA STRESS
Abstract
The activation energy of 13 kcal/M for loss of peripheral vision in man subjected to acceleration stress of +4 to +5 Gz resembles that of 12 kcal/M for survival of rats of +40 Gz, which suggests that the physiological mechanisms of acceleration protection are similar in the two species. The activation energy of survival to hypoxia stress (mouse) is 8.4 kcal/M which resembles the value of 8 kcal/M obtained for function of the normal human brain (alpha frequency of the EEG), which supports the concept that survival of the organism to hypoxia stress is indeed limited by survival of brain function. The large difference in activation energy of acceleration stress from that of hypoxia or of normal brain function suggests that tolerance to acceleration stress is not limited simply by the ability of nervous tissue to endure hypoxia, but must be dependent upon additional mechanisms.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 02, 1970
- Accession Number
- AD0709569
Entities
People
- Freeman W. Cope
Organizations
- Naval Air Warfare Center Warminster