Chloroamphenicol-Induced Hemolysis in Caucasian Glucose-6-Phosphate Dehydrogenase Deficiency

Abstract

Three white G6PD-deficient patients suffered severe hemolytic reactions during treatment for typhoid fever with chloramphenicol. Two of these patients were studied when free of infection to determine the hemolytic potential of chloramphenicol in the noninfected G6PD-deficient white person. It was found to be mildly hemolytic under these conditions, suggesting that a drug- disease synergism was primarily responsible for the clinical hemolytic reactions. The febrile state itself, or changes in plasma amino acids accompanying infection, may be responsible for disease-related hemolysis.

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Document Details

Document Type
Technical Report
Publication Date
Jun 29, 1971
Accession Number
AD0747137

Entities

People

  • Charles H. Halsted
  • Mohammed Fathy Abdel Wahab
  • R. P. Robertson
  • Ronald P. Mccaffrey

Organizations

  • Naval Medical Research Unit Three

Tags

DTIC Thesaurus Topics

  • Amino Acids
  • Anti-Bacterial Agents
  • Base Lines
  • Body Weight
  • Cell Physiological Processes
  • Cells
  • Deficiencies
  • Diseases And Disorders
  • Gram-Negative Bacterial Infections
  • Health Services
  • Hematocrit
  • Hemolysis
  • Hospitals
  • Infection
  • Internal Medicine
  • Navy
  • Wound Infections

Fields of Study

  • Biology
  • Medicine

Readers

  • Immunology
  • Parasitology and Pharmacology of Malaria.