Deiodination of L-thyroxine in vitro by Peripheral Leukocytes from Rhesus Monkeys with Bacterial Sepsis

Abstract

The deiodination of l-thyroxine (T4) in vitro by peripheral leukocytes isolated from healthy rhesus monkeys was compared to that of leukocytes from monkeys with acute Salmonella typhimurium sepsis, an infection associated with accelerated metabolism of T4 in vivo. Deiodination of T4 by leukocytes from septic monkey donors was significantly enhanced, with inorganic iodide identified chromatographically as the predominant product of T4 degradation. Induction of phagocytosis in vitro potentiated the T4 deiodinating activity of leukocytes from both control and infected monkeys. However, the proportion of added T4 degraded by leukocytes from septic donors following stimulation of phagocytosis in vitro was nearly twice that of cells from controls. The data suggest that deiodination of T4 by host neutrophils might contribute to the acceleration of T4 metabolism observed in vivo during some acute infections. (Modified author abstract)

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Document Details

Document Type
Technical Report
Publication Date
Mar 01, 1974
Accession Number
AD0784525

Entities

People

  • Frederick R. Derubertis

Organizations

  • United States Army Medical Research Institute of Infectious Diseases

Tags

DTIC Thesaurus Topics

  • Anti-Infective Agents
  • Bacteria
  • Biomedical Research
  • Cells
  • Clinical Medicine
  • Degradation
  • Health Services
  • Infection
  • Inoculation
  • Leukocytes
  • Materials
  • Metabolism
  • Monkeys
  • Rhesus Monkeys
  • Scintillation Counters
  • Thyroxine
  • Wound Infections

Fields of Study

  • Biology

Readers

  • Geochemistry
  • Immunology and Pathology
  • Microbial Pathology