Wnt/beta-Catenin, Foxa2, and CXCR4 Axis Controls Prostate Cancer Progression
Abstract
Wnt/beta-Catenin signaling and associated target genes are implicated in the establishment of bone metastasisand in the development of castration resistant prostate cancer. Our previous studies have shown that Foxa2 is aWnt/beta-catenin target gene in prostates. Our preliminary study suggests a WntFoxa2CXCR4 axis that isinvolved in PCa bone metastasis, and activation of this axis provides survival mechanisms for PCa cellsfollowing androgen deprivation. The hypothesis is that the Wnt/beta-catenin activation of Foxa2 and CXCR4promotes progression to CRPCa and facilitates bone colonization by PCa cells, and that targeting this axis willprovide a novel treatment for PCa bone metastasis and relapse after androgen ablation.Last October 1st, I moved from Vanderbilt to LSU Health Sciences Center, Shreveport, LA. This award isundergoing an institutional transfer. Since funds have been tied up during the transition, I have not been ableto conduct any research related to this project since I moved to LSUHSC.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2015
- Accession Number
- AD1000971
Entities
People
- Xiuping Yu
Organizations
- Vanderbilt University Medical Center