Scaffold Attachment Factor B1: A Novel Chromatin Regulator of Prostate Cancer Metabolism

Abstract

Progress was made in four major areas of study in the first year of the funding period. First, I determined the overlapping target genes of SAFB1 and AR. Second, I identified a SAFB1-dependent gene signature in clinical specimens using a large prostate cancer transcriptome dataset I assembled. Interestingly, the enriched cellular processes were associated with aggressive cancer behavior, including steroid hormone response, blood vessel formation, and RNA processing. Third, I found I could classify prostate cancer subtypes into three distinct categories, using only transcriptome data. One of these subtypes is functionally not possible to characterize, based on currently published profiling and genomic analyses. Fourth, I searched for possible regulators of this newly identified subtype using an integrative network analysis of hundreds of castration-resistant PC RNA expression profiles. This analysis revealed a set of key transcription factors (TFs) that appear to play a major role in metastatic castration-resistant prostate cancer (mCRPC). Among these TFs, we identified ONECUT2, which appears to be a master regulator of the novel prostate cancer subtype in our classification scheme.

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Document Details

Document Type
Technical Report
Publication Date
Aug 01, 2015
Accession Number
AD1007985

Entities

People

  • Jayoung Kim
  • Michael R Freeman
  • Sungyong You

Organizations

  • Cedars-Sinai Medical Center

Tags

DTIC Thesaurus Topics

  • Androgen Receptors
  • Blood
  • Blood Vessels
  • Computational Biology
  • Computational Science
  • Data Sets
  • Diseases And Disorders
  • Gene Expression
  • Health Services
  • Medical Personnel
  • Metabolism
  • Neoplasms
  • Polymerase Chain Reaction
  • Prostate Cancer
  • Proteins
  • Stem Cells
  • Transcription Factors

Fields of Study

  • Biology

Readers

  • Oncology (Cancer Research).
  • Oncology and Biomarker-Based Cancer Detection.
  • Prostate Cancer Biology.