Partial Complementation of the UV Sensitivity of Deinococcus Radiodurans Excision Repair Mutants by the Cloned denv Gene of Bacteriophage T4

Abstract

Deinococcus radiodurans has two endonucleases that incise UV irradiated DNA, UV endonuclease-alpha and UV endonuclease-beta, that are believed to functionally overlap. Both endonucleases must be mutationally inactivated to yield an incisionless, markedly UV-sensitive phenotype. denV, the bacteriophage T4 gene encoding pyrimidine dimer-DNA glycosylase (PD-glycosylase), was introduced and expressed via duplication insertion in D. radiodurans wild type, and single and double UV endonuclease mutants. Expression of denV increased UV survival of an incisionless double mutant and the strain deficient only in UV endonuclease-beta. The strain deficient in UV endonuclease-a has wild-type UV-resistance, and the expression of PD-glycosylase exerted no survival effect on this strain or wild-type. These results suggest that UV endonuclease-alpha does not recognize one or more types of cyclobutane dimer incised by the PD-glycosylase or UV endonuclease-beta

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Document Details

Document Type
Technical Report
Publication Date
Jul 19, 1990
Accession Number
AD1010984

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  • Hanlong Yao

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  • Uniformed Services University of the Health Sciences

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