Whole Exome Analysis of Early Onset Alzheimer's Disease

Abstract

The primary focus toward identification of Alzheimer disease (AD) risk genes over the past five years has been testing the common disease common variant (CDCV) hypothesis through the use of genome-wide association studies (GWAS) in late onset Alzheimer disease (LOAD). While common variation clearly plays a role in AD there is a growing realization that the CDCV hypothesis is unlikely to explain all the genetic effect underlying AD. One alternative hypothesis invokes multiple rare variants (RV) in one or more genes, each with stronger individual effects than CDCV genes. We designed this project to test the rare variant hypothesis in AD by examining those cases with the most severe phenotype as determine by early onset (EOAD, cases with AAO < 60 years). Although there are three known EOAD genes (PS1, PS2 and APP) they account for only ~60-70 of familial EOAD and even less of sporadic EOAD.

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Document Details

Document Type
Technical Report
Publication Date
Apr 01, 2016
Accession Number
AD1012967

Entities

People

  • Margaret A. Pericak-Vance

Organizations

  • University of Miami

Tags

DTIC Thesaurus Topics

  • African Americans
  • Alzheimer Disease
  • Biology
  • Cells
  • Computational Biology
  • Dementia
  • Demographic Cohorts
  • Genes
  • Genetic Phenomena
  • Genetic Structures
  • Genetics
  • Genome
  • Genotypes
  • Medical Genetics
  • Neurodegeneration
  • Neurons
  • Stem Cells

Fields of Study

  • Biology

Readers

  • Molecular and genetic basis of cancer.
  • Neurodegenerative Parkinson's Disease and Rickettsial Disease handbook, including the data level of dopamine, BC, neurons, and PD.

Technology Areas

  • Biotechnology