A New Paradigm for Ovarian Sex Cord-Stromal Tumor Development
Abstract
Transforming growth factor beta (TGFB) family members regulate multiple cellular functions and key reproductive processes in a contextually dependent manner via the interaction with membrane associated serine/threonine kinase receptor complexes (TGFBR1/TGFBR2) and downstream SMAD proteins. To complement our mouse model containing a constitutively active TGFBR1 using growth differentiation factor 9 (Gdf9)-Cre (i.e., TGFBR1-gCA), we herein generated a mouse model using Zp3-Cre line (termed TGFBR1-zCA). We performed a number of experiments including H and E staining, immunohistochemistry, and apoptosis assay to analyze potential ovarian phenotype of these mice. In contrast to TGFBR1-gCA mice, the TGFBR1-zCA mice did not develop ovarian tumors and demonstrated essentially normal ovarian histology and expression of granulosa cell and germ cell proteins. Using ovarian RNA from TGFBR1-gCA mice and controls, we performed RNA-seq experiment. Initial analysis identified 1301 genes that were differentially regulated in the TGFBR1-zCA ovaries versus controls. Interestingly, a number of genes are associated with folliculogenesis and oogenesis. Further studies will be focused on exploiting the RNAseq data and defining key regulators and/or pathways for ovarian tumor development.
Document Details
- Document Type
- Technical Report
- Publication Date
- May 01, 2017
- Accession Number
- AD1046813
Entities
People
- Qinglei Li
Organizations
- Texas A&M University