Metal Ion-Catalyzed Alcoholysis as a Strategy for the Destruction of Organophosphorus Chemical Warfare Agents

Abstract

Metal ion-catalyzed alcoholysis has proven to be an effective strategy for the rapid transformation of neutral reactive organophosphate esters of the phosphate, phosphonate, phosphonothioate, and phosphonothioate classes. This chemistry, using La3 (OMe)/methanol as a catalyst or solvent, which was applied to the V- and G-classes of chemical warfare agents, demonstrated extremely rapid transformation to low toxicity esters with load factors up to ~30 for nonfluoride-releasing agents. Variation of the alcohol solvent is tolerated. Variations of the metal catalyst provide potential redress to the fluoride inhibition of the class of G-agents. These observations indicate that formulations based on mixed alcohol solvents, combined with optimized metal catalysts for use in field decontamination, civilian security, and infrastructure scenarios, provide a pathway for tuning the reaction media while retaining rapid destruction kinetics.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2018
Accession Number
AD1055800

Entities

People

  • Alexei A. Neverov
  • Andrea Tamer
  • Frederic J. Berg
  • H. Dupont Durst
  • R. S. Brown

Organizations

  • Edgewood Chemical Biological Center

Tags

Communities of Interest

  • Advanced Electronics
  • Counter WMD
  • Human Systems
  • Weapons Technologies

DTIC Thesaurus Topics

  • Alcohols
  • Catalysts
  • Chemical Analysis
  • Chemical Reactions
  • Chemical Synthesis
  • Chemical Warfare
  • Chemical Warfare Agents
  • Chemical Weapons
  • Chemistry
  • G Agents
  • Gd Agent
  • Liquid Chromatography
  • Magnetic Resonance
  • Mass Spectrometry
  • Nerve Agents
  • Nuclear Magnetic Resonance
  • Organic Chemistry
  • Organophosphates
  • Organophosphorus Compounds
  • Phosphorus
  • Resonance
  • Security
  • Spectrometry
  • Spectroscopy

Readers

  • Electrochemical Engineering/ Fuel Cell Technologies
  • Environmental Engineering.
  • Neurotoxicology