Characterizing Myeloid Cell Activation in NF1 Vasculopathy

Abstract

Our original proposal seeks to understand the mechanisms underlying macrophage recruitment and activation in NF1-related vasculopathy. We have developed two mouse models that closely recapitulate NF1-related arterial stenosis and aortic aneurysm formation and propose to examine the contribution of MCP-1/CCR2 signaling and redox biology to these pathologies. As aneurysm and arterial stenosis represent two ends of a continuum, perturbations in macrophage recruitment and redox biology may underlie both pathologies; therefore, we have attempted to take a broad approach to both models in regards to these mechanisms. We have completed all proposed experiments, which have been described in several high-impact publications with multiple manuscripts undergoing Editorial Review for publication.

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Document Details

Document Type
Technical Report
Publication Date
Mar 01, 2019
Accession Number
AD1096040

Entities

People

  • Brian K. Stansfield

Organizations

  • Augusta University

Tags

DTIC Thesaurus Topics

  • Aneurysm
  • Cardiovascular Physiological Phenomena
  • Cardiovascular System
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Health Services
  • Medical Personnel

Fields of Study

  • Biology
  • Medicine

Readers

  • Cardiovascular Physiology
  • Immunology and Pathology
  • Neuroscience