Therapeutic Targeting of Disrupted Nucleocytoplasmic Transport in C9orf72-ALS Motor Neurons
Abstract
Our proposed therapeutic development strategy is to carry out a screening campaign in order to identify small molecules that restore nucleocytoplasmic transport in C9ORF72 related amyotrophic lateral sclerosis. We developed a cell-based screening assay using nucleocytoplasmic transport biosensor. The sub-cellular localization of the biosensor can be assessed using high content imaging. In the second year of funding we began screening all compound libraries but encountered problems that substantially slowed our progress. We successfully overcame these problems and have now completed the primary screen of all libraries. However, due to the issues with quality control, we fell behind schedule and requested a no cost extension to allow us more time to complete the final validation experiments. The extension was approved and we finished the project by validating hits in U-2OS cells and iPSC neurons.
Document Details
- Document Type
- Technical Report
- Publication Date
- Aug 01, 2020
- Accession Number
- AD1110710
Entities
People
- Zane Zeier
Organizations
- University of Miami