Posttranscriptional Regulation of Inflammatory Molecules in Cystic Fibrosis

Abstract

Cystic fibrosis (CF) is an autosomal recessive disorder caused by mutations in a chloride ion channel called cystic fibrosis transmembrane conductance regulator (CFTR). CF patients frequently develop chronic lung inflammation and refractory respiratory infection, most commonly by Pseudomonas aeruginosa (P. aeruginosa, PA). The CF lung disease is one of the major causes of the morbidity and mortality in CF patients, and development of anti-inflammatory therapeutics for CF is a major goal. Inflammatory molecules are regulated through posttranscriptional mechanisms by microRNAs (miRNAs) and mRNA-binding proteins. miRNAs are endogenous non-coding RNA molecules that have been found to regulate various cellular processes by destabilizing target mRNAs. Recent studies indicate that microRNAs are key molecules in disease development and therapy. Additionally, several RNA-binding proteins (RBP) have been shown to coordinate with miRNAs and regulate expression of inflammatory genes. A class of RBPs recognize and bind to adenosine-uridine rich regions (ARE regions) in the 3 UTRs of mRNAs and are known as ARE-binding proteins (AUBPs). However, the role of these factors in the regulation of the inflammation in CF has not been well studied. Therefore, my overall research theme is to determine posttranscriptional regulation of inflammatory molecules in CF. Based on previous findings from our laboratory, my thesis especially focuses on the mechanisms of posttranscriptional regulation of IL-8 expression in CF by miR-155 and HuR. Our findings indicated that inflammatory mediators and Pseudomonas infection modulate IL-8 and as well as miR-155 expression in CF lung epithelial cell. To further investigate mechanisms of posttranscriptional regulation of IL-8 mRNA, we subsequently searched for proteins and miRNAs which bind to the ARE region of IL-8 mRNA specifically in CF. Our data show that HuR and miR-16 bind to the ARE region, and regulate IL-8 mRNA stability. Additionally we examined

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Document Details

Document Type
Technical Report
Publication Date
Sep 10, 2015
Accession Number
AD1127928

Entities

People

  • Motohiro Tsuchiya

Organizations

  • Uniformed Services University of the Health Sciences

Tags

DTIC Thesaurus Topics

  • Bacteria
  • Bacterial Infections
  • Bacterial Proteins
  • Biological Factors
  • Biomedical And Dental Materials
  • Blood
  • Breast Cancer
  • Cell Physiological Processes
  • Cells
  • Cellular Structures
  • Chemical Synthesis
  • Chemistry
  • Epithelial Cells
  • Gene Expression
  • Genetics
  • Lung Diseases
  • Molecular Biology
  • Peptide Growth Factors
  • Peptides
  • Proteins
  • Rna Stability

Fields of Study

  • Biology

Readers

  • Immunology and Pathology
  • Microbial Pathology
  • Molecular Genetics