The Epigenetic Adaptation of ER+ Breast Cancer Cells to the Bone Microenvironment

Abstract

Bone metastases more frequently occur to estrogen receptor positive (ER+) breast cancer. Thus, it is imperative to understand how disseminated ER+ cancer cells escape therapies in distant organs and to identify therapies that can eliminate these cells. Supported by the DoD Era of Hope Award, my laboratory developed a series of models and techniques to investigate cancer-bone interaction at a single cell resolution. We have also elucidated an epigenomic reprogramming effect elicited by the bone microenvironment that leads to endocrine resistance. Importantly, this represents a mechanism fundamentally distinct from classic clonal evolution process. In this application, we aim to expand our research to understand the epigenetic mechanisms underlying the above mentioned effects. More importantly, we wish to identify therapeutic strategies that can reverse the endocrine resistance and prevent recurrences of ER+ breast cancer. It is important to note that multiple lines of evidence have emerged and suggest that many metastases are actually seeded by other metastases, rather than primary tumors. Two thirds of bone-only metastases are followed by appearance of metastases in multiple other organs. Our unpublished studies also demonstrated that bone microenvironment may invigorate disseminated breast cancer cells for further metastasis. Thus, our research focusing on bone metastasis may indeed generate broader impact and lead to reduction of metastases in other organs as well. Taken together, the proposed research represents a move beyond incremental advancement, and will likely lead to new metastatic and adjuvant therapies. Our study will demonstrate how epigenomic adaptation drives cancer evolution and alter therapeutic responses together with clonal selection, provides insights into the clinical enigma of ER+ metastatic recurrences despite endocrine therapies, and leads to reduction of metastasis-related breast cancer mortality.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2021
Accession Number
AD1147536

Entities

People

  • Xiang Zhang

Organizations

  • Baylor College of Medicine

Tags

DTIC Thesaurus Topics

  • Arteries
  • Bone And Bones
  • Bone Diseases
  • Breast Cancer
  • Cell Movement
  • Cell Physiological Processes
  • Cells
  • Cellular Structures
  • Chemistry
  • Culture Techniques
  • Gene Expression
  • Genetics
  • Health Services
  • Information Science
  • Intercellular Junctions
  • Peptide Growth Factors
  • Prostheses And Implants
  • Proteins
  • Statistical Analysis
  • Surgery
  • X-Ray Computed Tomography

Fields of Study

  • Biology

Readers

  • Breast cancer cell signaling and growth regulation.
  • Oncology (Cancer Research).
  • Systems Analysis and Design