Longitudinal Analysis of Disease-Site Activities Impairing Wound Healing in Epidermolysis Bullosa and Development of Therapeutic Strategies

Abstract

Epidermolysis bullosa patients develop poorly healing skin wounds that are frequently colonized with microbiota. To evaluate the dynamics of the microbiota colonizing early, established, and chronic EB wounds, we conducted high throughput sequencing to define multiple metrics of the microbiome, including diversity, stability, and relative abundance of potential pathogens and identified mirobiomic features associated with disease clinical outcomes. We found that progression of RDEB wounds to chronic state is associated with reduced abundance of specific taxa and overall reduced diversity of bacterial communities. Specifically, it is characterized by disappearance of Corynebacterium, Propionobacterium, and several other taxa and accumulation of pre-dominantly Staphylococcus and Pseudomonas species in chronic wounds. Because T cells play an important role in clearing such pathogens, we defined the status of adaptive T cell-mediated immunity in wounds. We found that RDEB wounds and epithelial cells are most frequently infected with Staphylococcus sp. and Pseudomonas sp. The wound-associated T cells contain populations of CD4+ and CD8+ peripheral memory T cells that respond to soluble microbial antigens by proliferating and secreting interferon gamma (IFNgamma). Also, CD8+ cytotoxic T lymphocytes recognize S. aureus-infected RDEB keratinocytes and respond by producing interleukin-2 (IL-2) and IFNgamma and degranulating and cytotoxically killing infected cells. Prolonged exposure of RDEB-derived T cells to microbial antigens in vitro does not trigger PD1-mediated T cell exhaustion but induces differentiation of the CD4high population into CD4highCD25+FoxP3+ regulatory T cells. Collectively, our data demonstrated that adaptive T cell-mediated immunity could clear infected cells from wound sites, but these effects might be inhibited by PD1/Treg-mediated immuno-suppression.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 2021
Accession Number
AD1155394

Entities

People

  • Jouni Uitto
  • Olga Igoucheva
  • Vitali Y Alexeev

Organizations

  • Thomas Jefferson University

Tags

DTIC Thesaurus Topics

  • Adaptive Immunity
  • Bacteria
  • Bacterial Infections
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Culture Media
  • Diseases And Disorders
  • Epithelial Cells
  • Foot Diseases
  • Health Services
  • Immune System
  • Immunity
  • Infection
  • Lymphocytes
  • Medical Personnel
  • Microbiomes
  • Polymerase Chain Reaction
  • Skin Diseases
  • Staphylococcus Aureus

Fields of Study

  • Biology
  • Medicine

Readers

  • Immunology and Pathology
  • Microbial Pathology
  • Trauma Surgery or Emergency Medicine.

Technology Areas

  • Biotechnology
  • Biotechnology - Cancer Biotech