Transcription Factor Analysis of SLE

Abstract

Purpose: Define transcription factors critical for SLE pathogenesis Scope:1) Expand chromatin marks to identify latent enhancers and regulatory regions relevant for SLE 2) Inhibit identified TF to define cellular effects. A key aspect was comparing different clinical subsets of SLE and using patients with rheumatoid arthritis as inflammatory controls. Major findings: Due to the pandemic, we only began our wet bench library preparation in January. We also suffered form the same pipeline supply chain issues as most molecular biology labs. Although we have data, we have not done any analysis.

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Document Details

Document Type
Technical Report
Publication Date
Oct 01, 2021
Accession Number
AD1165682

Entities

People

  • Kathleen E. Sullivan

Organizations

  • Johns Hopkins School of Medicine

Tags

DTIC Thesaurus Topics

  • Arthritis
  • Autoimmune Diseases
  • Biomedical Research
  • Blood
  • Cells
  • Connective Tissue Diseases
  • Data Analysis
  • Department Of Defense
  • Disease Attributes
  • Diseases And Disorders
  • Epigenetics
  • Factor Analysis
  • Genetics
  • Health Services
  • Immune System
  • Kidney Diseases
  • Lupus
  • Medical Personnel
  • Molecular Biology
  • Proteins
  • Supply Chain
  • Transcription Factors

Fields of Study

  • Medicine

Readers

  • Clinical Trial Research.
  • Molecular Biology and Genetics
  • Neurological Diseases/Conditions/Disorders