Gene Expression in Blood, Lung, and Heart Following Exposure of Rats to Hypobaria

Abstract

Results obtained from rodent traumatic brain injury models indicate that exposure to air-evacuation-relevant hypobaria within a few days following injury worsens neurologic outcomes and exacerbates inflammatory damage to the brain, lungs, and heart. The causes for these changes are not well understood but could involve changes in gene expression in response to moderate hypobaria and hyperoxia, which often occur during air evacuation. This study tested the hypothesis that exposure of normal healthy rats to hypobaria (4000 or 8000 ft altitude) for as short as 5 hr and as long as 10 hr results in tissue-selective changes in gene expression that could provide insight into the mechanisms responsible for these effects.

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Document Details

Document Type
Technical Report
Publication Date
Mar 10, 2022
Accession Number
AD1170340

Entities

People

  • Gary Fiskum

Organizations

  • University of Maryland School of Medicine

Tags

DTIC Thesaurus Topics

  • Acids
  • Aeromedical Evacuation
  • Air Force
  • Air Force Research Laboratories
  • Brain Injuries
  • Cell Physiological Processes
  • Chemical Reactions
  • Chemistry
  • Combat Injuries
  • Deoxyribonucleic Acids
  • Department Of Defense
  • Gene Expression
  • Governments
  • Health Services
  • High Altitude
  • Inflammation
  • Medical Personnel
  • Microarray Analysis
  • Oxidative Stress
  • Patient Care
  • Ribonucleic Acids
  • Transcriptomics

Fields of Study

  • Biology

Readers

  • Cardiovascular Physiology
  • Molecular and genetic basis of cancer.
  • Trauma or Military Medicine