Developing Strategies to Overcome Prostate Stromal-Derived NRG1-Mediated Resistance to AR Blockade in High-Risk Locally Advanced Prostate Cancer

Abstract

Through pre-clinical studies, we have recently discovered for the first time, that the prostate cancer microenvironment stromal cells, secrete neuregulin (NRG1) in response to androgen blockade, which promotes the cancer cells to survival an androgen deprived state. Our recent finding explains in part why, so few high-risk primary prostate cancers achieve a complete pathologic response to androgen blockade. Importantly, the neuregulin signaling pathway is therapeutically actionable with several clinical agents either FDA approved for other malignancies or in early development. Our research proposal aims to define the downstream mechanisms through which NRG1 promotes cell survival and determine the therapeutic strategies targeting the NRG1 pathway which enhance response to androgen blockade. Our work has the potential to improve our understanding of how the cancer environment influences resistance to androgen blockade and lead to novel combination therapies vastly changing the standard of care for men with high-risk prostate cancer.

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Document Details

Document Type
Technical Report
Publication Date
Jul 01, 2023
Accession Number
AD1205238

Entities

People

  • Brett S. Carver

Organizations

  • Memorial Sloan Kettering Cancer Center

Tags

DTIC Thesaurus Topics

  • Androgen Receptors
  • Androgens
  • Biomedical Research
  • Bladder Cancer
  • Cancer
  • Cell Physiological Processes
  • Cells
  • Clinical Trials
  • Combination Therapy
  • Department Of Defense
  • Diseases
  • Indirect Costs
  • Medical Personnel
  • Neoplasms
  • Oncology
  • Prostate Cancer
  • Resistance
  • Standards
  • Stromal Cells
  • Therapy

Fields of Study

  • Medicine

Readers

  • Immunology and Pathology
  • Oncology (Cancer Research).
  • Prostate Cancer Biology.