Longitudinal Analysis of Disease Site Activities Impairing Wound Healing in Epidermolysis Bullosa and Development of Therapeutic Strategies

Abstract

Hereditary Epidermolysis Bullosa (EB) is a mechanobullous disease characterized by the fragility of the skin. Separation of skin layers, a hallmark of the disease, leads to the development of skin lesions ranging from skin blisters to chronic ulcerated wounds, which represent the major health-related burden for EB patients as these lesions are associated with infection,sepsis, dehydration, deformities, and cancer. Our previous findings along with several clinical observations strongly suggest that detailed characterization of molecular and cellular events impairing wound healing is crucial to improve stem cell-based and combination treatment modalities. The mechanisms that control the fate of the EB wounds remain poorly understood. Our overarching hypothesis is that an altered secretome along with microbial infection at EB wounds lead to the establishment of pro-inflammatory feedback loops and inhibition of wound healing and that targeting of chronic skin lesions with pharmacological treatments can restore aberrant wound healing and skin integrity. These questions were tested by investigating molecular and cellular mechanisms underlying unresolved wound healing, characterizing microbial dynamics and immunogenicity of the wound colonizing bacteria, and developing pharmacological approaches for EB treatment and management. By using samples from three major EB types, we identified type-specific changes in wound healing. We also delineated cellular populations and pathways preventing wound healing that were amenable for therapy development. In conjunction with state-of-the-art microbiome sampling and bio-informatics, our studies accelerated understanding of the differences between chronic and healing wounds. Conducted analysis of wound progression helped to define precipitating factors that predefine EB wound aberrations.

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Document Details

Document Type
Technical Report
Publication Date
Dec 01, 2022
Accession Number
AD1212326

Entities

People

  • Olga Igoucheva

Organizations

  • Thomas Jefferson University

Tags

DTIC Thesaurus Topics

  • Acquisition
  • Adaptive Immunity
  • Antigens
  • Bacteria
  • Bacterial Infections
  • Cell Physiological Processes
  • Cells
  • Diseases
  • Epithelial Cells
  • Foot Diseases
  • Granulocytes
  • Health Services
  • Infection
  • Lymphocytes
  • Medical Personnel
  • Microbiomes
  • Skin Diseases
  • Wound Healing
  • Wound Infections

Fields of Study

  • Biology

Readers

  • Immunology and Pathology
  • Molecular and genetic basis of cancer.
  • Trauma Surgery or Emergency Medicine.

Technology Areas

  • Biotechnology