Mechanisms of Metastasis Suppression and Translational Applications in Thyroid Cancer
Abstract
Progressive metastases are responsible for most deaths from thyroid cancer. We have identified that loss of RCAN1.4 is a key event in metastatic progression due to increased NFE2L3-mediated IL-8 expression and by inducing a tumor-promoting immune environment. The IL-8 system is not able to be studied in mouse models and the pace of translation to define regulatory mechanisms regulating metastatic progression in immune competent systems requires arduous mouse models that delay translational efficiency. To test the role of IL-8 and RCAN1.4 loss and to model the tumor: immune interface in a human cell based model with more rapid translational potential, we will develop a highly reproducible thyroid cancer metastasis on a chip platform that will enable mechanistic studies of the RCAN 1.4 metastasis suppression pathway, create a new platform to study mechanistic regulation of the tumor-immune interface, and allow for more rapid validation of biomarkers and therapeutic targets.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 01, 2023
- Accession Number
- AD1212988
Entities
People
- Aleksander Skardal
- Matthew D Ringel
Organizations
- Ohio State University