Hepatic Pathology in Mice after Continuous Inhalation Exposure to 1,1,1- Trichloroethane

Abstract

Male CF-1 mice (24-34g) were exposed to either 250ppm or 1,000ppm 1, 1,1-trichloroethane in air continuously for 14 weeks. Control mice were exposed to room air. Serial sacrifice of exposed and control mice from 1 to 14 weeks demonstrated significant changes in the centrilobular hepatocytes of animals in the 1,000ppm group. Moderate liver triglyceride accumulation was evident in the 1,000ppm group and peaked at 40mg/gm of tissue (wet weight) after 7 weeks of exposure. Partial recovery was indicated by a drop in the hepatic triglyceride level to 16mg/gm by 14 weeks of exposure to 1,000ppm. Electron microscopy revealed that cytoplasmic alterations were most severe in centrilobular hepatocytes in the 1,000ppm group, and were mild to minimal in the 250ppm group. These alterations consisted of vesticulation of the rough endoplasmic reticulum with loss of attached polyribosomes, increased smooth endoplasmic reticulum, microbodies, and triglyceride droplets.

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Document Details

Document Type
Technical Report
Publication Date
Aug 01, 1975
Accession Number
ADA020124

Entities

People

  • Ernest E. Mcconnell
  • Fred Morris
  • Neil S. Mcnutt
  • Robert L. Amster

Organizations

  • Air Force Research Laboratory

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Air Force
  • Air Force Facilities
  • Alkanes
  • Biomedical Research
  • Carbon Tetrachloride
  • Cells
  • Cellular Structures
  • Cytoplasmic Granules
  • Cytoplasmic Vesicles
  • Electron Microscopy
  • Endoplasmic Reticulum
  • Fatty Acids
  • Governments
  • Laboratory Animals
  • Manned Spacecraft
  • Organelles
  • Rodents

Readers

  • Toxicology/Environmental Toxicology

Technology Areas

  • Microelectronics
  • Microelectronics - Graphene