Pulmonary Response to Hemorrhagic Shock.

Abstract

Positive end expiratory pressure has been found to (1) alter pulmonary metabolism activity because of increased lung stretch; (2) cause the release of a humoral agent which gains access to the systemic and coronary circulation; (3) produce a decrease in left ventricular contractility and cardiac output. These findings are reported in both dogs and patients suffering acute respiratory failure. The entrapment of granulocytes and platelets has been demonstrated in animals and man with respiratory failure. (Author)

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Document Details

Document Type
Technical Report
Publication Date
Apr 30, 1977
Accession Number
ADA045823

Entities

People

  • Herbert B. Hechtman

Organizations

  • Boston University

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Aneurysm
  • Animals
  • Arteries
  • Biomedical Research
  • Birds
  • Blood
  • Cardiovascular Physiological Phenomena
  • Cardiovascular System
  • Cells
  • Granulocytes
  • Health Services
  • Hemorrhagic Shock
  • Medical Personnel
  • Metabolism
  • X Rays

Fields of Study

  • Medicine

Readers

  • Cardiovascular Physiology