'Escherichia coli' Shock Following Corticosteroid Treatment: A Pathologic Study in Baboons.

Abstract

The purpose of this study was to determine if corticosteroids would prevent the development of the pathologic lesions of septic shock in baboons treated with live E. coli organisms during a 24-hour study period. Pathologic changes were defined for multiple organs and compared to the lesions previously described in shock in humans. Eleven awake baboons were infused with comparable doses of live E. coli organisms during a 5-hour period with one member of each pair receiving methylprednisolone. One additional animal received saline in place of organisms and served as a control. Results of this study would indicate that adrenal corticosteroids do not prevent and/or decrease the severity of any of the morphologic lesions in the baboon subjected to low or high doses of E. coli organisms. The pathologic changes induced by live E. coli organisms in the baboon include fibrin thrombi, edema and/or hemorrhage, and necrosis of multiple organ systems. The liver, adrenal, and kidney show striking pathologic changes whereas the gastrointestinal tract shows no significant pathologic alteration. This morphologic study demonstrates that the shock lesions of the subhuman primate and human are remarkably similar.

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Document Details

Document Type
Technical Report
Publication Date
Jun 12, 1978
Accession Number
ADA057458

Entities

People

  • B. A. Benjamin
  • B. K. Beller
  • J. J. Coalson
  • Linda T. Archer
  • R. H. Spaet

Organizations

  • University of Oklahoma Health Sciences Center

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Animals
  • Arteries
  • Biological Sciences
  • Biomedical Research
  • Blood
  • Blood Cells
  • Blood Vessels
  • Capillary Electrophoresis
  • Cardiovascular System
  • Cells
  • Electron Microscopes
  • Electron Microscopy
  • Escherichia Coli
  • Hemorrhage
  • Hemorrhagic Shock
  • Military Research
  • United States

Fields of Study

  • Medicine

Readers

  • Cardiovascular Physiology
  • Toxicology/Environmental Toxicology