Myocardial Function in Endotoxin Shock.

Abstract

Coronary hypoperfusion and depressed responses to beta adrenergic stimuli are prime factors in the elicitation of cardiac dysfunction. Intracardiac disturbances perform a major role but their causes are obscure. Associated together in this latter category are the development of edema in the myocardium occurring in both the contractile elements and mitochondria; ionic imbalances of potassium and probably calcium; and elevations of left ventricular end diastolic pressure and depressions of negative dP/dt sub max, cardiac power and efficiency. The interactions and additive effects of these factors are responsible for the development of myocardial dysfunction in endotoxin shock. (Author)

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Document Details

Document Type
Technical Report
Publication Date
Aug 28, 1978
Accession Number
ADA061525

Entities

People

  • Lerner B. Hinshaw

Organizations

  • University of Oklahoma Health Sciences Center

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Arteries
  • Biological Sciences
  • Biomedical Research
  • Blood
  • Blood Flow
  • Cardiovascular Diseases
  • Cardiovascular Physiological Phenomena
  • Cardiovascular System
  • Chemistry
  • Heart
  • Hemorrhagic Shock
  • Military Research
  • North Carolina
  • Oklahoma
  • Peptides
  • United States

Fields of Study

  • Biology
  • Medicine

Readers

  • Cardiovascular Physiology