The Action of Botulinum Toxin at the Neuromuscular Junction

Abstract

Botulism results from the action is of a protein neurotoxin (MW approx. 150,000) produced by the bacterium Clostridium botulinum, of which there are eight known strains. Botulinum neurotoxin is the most potent biological toxin known, having a median lethal dose of 5-50 ng/kg body weight. The primary site of action of botulinum toxin is the cholinergic nerve terminal, where it blocks the release of the neurotransmitter acetylcholine. Death usually results from respiratory failure. Nonlethal doses of botulinum toxin can induce sprouting of the nerve terminal and have significant postsynaptic effects, including muscle atrophy and alterations in the membrane electrical properties of the muscle fiber. There is no universally available treatment for botulinum intoxication. However, immunotherapeutic and chemotherapeutic procedures are now being developed and will be discussed.

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Document Details

Document Type
Technical Report
Publication Date
Dec 22, 1980
Accession Number
ADA094643

Entities

People

  • Lawrence C. Sellin

Organizations

  • United States Army Medical Research Institute of Infectious Diseases

Tags

DTIC Thesaurus Topics

  • Amines
  • Biomedical Research
  • Cell Physiological Processes
  • Cells
  • Cholinergic Nerves
  • Cranial Nerves
  • Death
  • Immunoglobulins
  • Membrane Potentials
  • Muscle Fibers
  • Neuromuscular Transmission
  • Neurotransmitters
  • Peripheral Nervous System
  • Skeletal Muscle
  • Spiders
  • Synapses
  • United States

Fields of Study

  • Biology

Readers

  • Microbial Pathology
  • Neuroscience
  • Toxicology/Environmental Toxicology