Chemical Carcinogen-Induced Changes in tRNA Metabolism in Human Cells

Abstract

It was proposed that changes in tRNA metabolism are required for cells to progress through the stages of carcinogenesis, and a comprehensive hypothesis was formulated to describe tRNA-mediated endogenous promotion of carcinogenesis. This hypothesis offers a viable explanation for the lengthy time frame observed between carcinogen exposure and neoplastic transformation. A role was defined for 7-methylguanine as an endogenous promoting agent, whereby this natural RNA catabolite induces queuine hypomodification in the tRNA anticodon by inhibiting the queuine insertion enzyme tRNA-guanine ribosyltransferase. Subsequently, 7-methylguanine induces neoplastic transformation. A cell culture system was developed which allows the study of tumor promoter-induced mimicry of transformation with normal human cells, and using this system, phorbol ester tumor promoters were also demonstrated to induce queuine hypomodification of TRNA. However, in this case, the hypomodification occurred due to a specific phorbol ester inhibition of queuine transport into the cells. Most importantly, overcoming the tumor promoter-induced hypomodification of tRNA by supplying the cells with excess queuine, blocked the expression of a transformed phenotype by the human cells. Therefore, queuine may be an anti-promoting compound, and a role for queuine hypomodification in the expression (promotion) of carcinogenesis appears likely.

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Document Details

Document Type
Technical Report
Publication Date
Nov 20, 1984
Accession Number
ADA150962

Entities

People

  • R. W. Trewyn

Organizations

  • Ohio State University

Tags

DTIC Thesaurus Topics

  • Cell Membrane
  • Cell Physiological Processes
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Health Services
  • Liquid Chromatography
  • Medical Personnel
  • Nucleosides
  • Organic Chemistry

Fields of Study

  • Biology

Readers

  • Computer Engineering
  • Molecular and genetic basis of cancer.
  • Oncology (Cancer Research).