A Program for Clinical Care in Physical Trauma.
Abstract
A septic sheep model has been developed which is similar to extensive cellulitis secondary to penetrating injury of the leg. This model increases the release of skeletal muscle amino acids, fails to develop ketosis during starvation and injury/infection, and appears suitable to evaluate therapies that would reverse the catabolic effects of injury. Protein turnover, determined in trauma-septic patients using stable isotopes, is increased. The increase in breakdown exceeds synthesis, and these alterations in amino acid metabolism account for the negative nitrogen balance following injury. Keywords: exogenous substrate; catabolic response.
Document Details
- Document Type
- Technical Report
- Publication Date
- Aug 01, 1981
- Accession Number
- ADA157300
Entities
People
- D. W. Wilmore
Organizations
- Harvard University