Gluatamic Acid Decarboxylase Activity Decreases in Mouse Neocortex after Lesions of the Basal Forebrain.

Abstract

Glutamic acid decarboxylase (GAD) activity was measured in the cerebral cortex of animals after acute and chronic lesions to basal forebrain cholinergic markers in parietal cerebral cortex. A statistically significant 30% decrease in GAD activity was first detected at 6 weeks postlesion and was still measurable 8 months after the lesion. These results suggest that cholinergic inputs to cortex indirectly or directly influence GABAergic transmission in cortex.

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Document Details

Document Type
Technical Report
Publication Date
Dec 22, 1986
Accession Number
ADA175700

Entities

People

  • C. F. Hohmann
  • F. F. Ebner
  • M. F. Bear

Organizations

  • Brown University

Tags

Communities of Interest

  • Materials and Manufacturing Processes

DTIC Thesaurus Topics

  • Alzheimer Disease
  • Brain
  • Brain Stem
  • Cerebral Cortex
  • Dementia
  • Diseases And Disorders
  • Glutamic Acid
  • Medical Personnel
  • Nervous System
  • Neurons
  • Neurosciences
  • Parkinson'S Disease
  • United States
  • United States Government

Fields of Study

  • Biology
  • Psychology

Readers

  • Neuroscience