Pharmacological Protection of the Retina against Damaging Laser Exposures: A Feasibility Study

Abstract

Light damage to ocular tissues may occur through at least three mechanisms -- thermal, photochemical, or mechanical (photoacoustic) stress. These stress mechanisms may act singly, or in combination, depending on the intensity, wavelength, and temporal parameters of the damaging light exposure. Because some of these mechanisms involve biochemical intermediates, the possibility exists that appropriate pharmacological therapy might limit the extent of retinal light damage. This investigation determined the degree to which pharmacological pretreatments could protect a model retina against laser light damage. Five agents were examined for protective activity: an anti-inflammatory steroid hormone (dexamethasone); three antioxidants (3-aminotyrosine, vitamins C AND E); and a chelating agent (EGTA). Antioxidant; Laser; Light damage; Protection; Rabbit; Retina.

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Document Details

Document Type
Technical Report
Publication Date
Sep 01, 1988
Accession Number
ADA201898

Entities

People

  • Randolph D. Glickman
  • Robert M. Cartledge
  • William R. Elliott Iii

Tags

Communities of Interest

  • Advanced Electronics
  • Human Systems
  • Materials and Manufacturing Processes
  • Space

DTIC Thesaurus Topics

  • Amplitude
  • Antioxidants
  • Cellular Structures
  • Chelate Compounds
  • Classification
  • Eye
  • Free Radicals
  • Heat Energy
  • Intensity
  • Ion Lasers
  • Krypton Lasers
  • Laser Safety
  • Lasers
  • Light Sources
  • Oxygen
  • Vitamin C
  • Vitamin E

Fields of Study

  • Biology

Readers

  • Cardiovascular Physiology
  • Surface Coatings Technology.
  • Vision Science/Vision Psychology/Cognitive Neuroscience.

Technology Areas

  • Directed Energy