Induction of Marrow Hypoxia by Radioprotective Agents

Abstract

Many compounds which possess sulfhydryl groups have been shown to protect bone marrow from radiation injury. The most effective thiol radioprotective agent is ethiofos (S-2-(3-aminopropylamino)ethylphosphorothoic acid or WR-2721). The ability of thiol and non-thiol radioprotectors to induce hypoxia was determined using binding of (3H)misonidazole by bone marrow cells as a measure of hypoxia. When administered at maximally radioprotective doses, four drugs (WR-2721, cysteamine, 5-hydroxytryptamine, and 16,16-dimethyl prostaglandin E2) significantly increased the amount of (3H)misonidazole bound by marrow cells, while no significant increase in binding was observed with three other agents (endotoxin, AET, superoxide dimutase). Doses of WR-2721 previously shown to provide suboptimal radioprotection did not significantly increase 3H-misonidazole binding. These results suggest that the physiological effects of some radioprotectors, that is, their ability to induce marrow hypoxia, may contribute to their efficacy in vivo. Keywords: Radio protective agents; Reprints.

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Document Details

Document Type
Technical Report
Publication Date
Jan 01, 1989
Accession Number
ADA214330

Entities

People

  • Cheryl Sawich
  • M. J. Allalunis-turner
  • Thomas L. Walden Jr.

Organizations

  • Armed Forces Radiobiology Research Institute

Tags

DTIC Thesaurus Topics

  • Blood
  • Blood Flow
  • Body Weight
  • Bone Marrow
  • Bone Marrow Cells
  • Bones
  • Cell Line
  • Cells
  • Chemistry
  • Free Radicals
  • Ionizing Radiation
  • Oxygen
  • Physiological Effects
  • Radiation
  • Radiation Injuries
  • Radiation Protection
  • Radioprotective Agents

Fields of Study

  • Biology
  • Medicine

Readers

  • Auditory Neuroscience/Auditory Physiology.
  • Molecular and Cellular Biochemistry
  • Toxicology/Environmental Toxicology