An Experimental Brain Missile Wound: Ascertaining Pathophysiology and Evaluating Treatments to Lower Mortality and Morbidity
Abstract
Chemical regulation of CBF: Normoxic hypocapnia (PaCO to the second power 21 mmHg) significantly reduced total CBF and many rCBFs both before and after wounding but after wounding this response may have been attenuated except around the wound track where CBF reduction was enhanced. Effect of wounding on selected physiologic variables which might account for death. The effect of brain wounding on lung water was insignificant. The effect increased intracranial pressure on brain biogenic amines. The monoaminergic neurotransmitters (EPI, NE, 5-HT) and some their metabolities (DOPAC, HVA, 5-HIAA) were evaluated in selected brain stem areas and the hypothalamus after increases in ICP sufficient to elicit an immediate Cushing response.
Document Details
- Document Type
- Technical Report
- Publication Date
- Oct 26, 1990
- Accession Number
- ADA230062
Entities
People
- Dan Torbati
- J. B. Farrell
- Joweph Soblosky
- June Davidson
- Michael E. Carey
Organizations
- LSU Health Sciences Center New Orleans