Free Radical Mechanisms of Xenobiotic Mammalian Cytotoxicities

Abstract

Our initial goal was to identify if free radical mechanisms are involved in the cytotoxicity of a number of IRP volume I and II chemicals. We found that a number of these agents act to enhance membrane lipid peroxidation in response to a standard dose of exogenous free radicals. Using chlorinated hydrocarbons (carbon tetrachloride, trichloroethylene, dichloroethylene, trichloroethane, dichloroethane) as a model for other IRP chemicals, we established conditions to measure lipid peroxidation in cultured smooth muscle and endothelial cells. These agents induced lipid peroxidation in the presence of physiological levels of iron in these vascular cells by a mechanism that doesn't require cytochrome P-450. Antiradical treatment with deferoxamine and probucol (but not SOD, catalase, or mannitol) appear to reduce the toxicity of these agents.

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Document Details

Document Type
Technical Report
Publication Date
Jun 30, 1991
Accession Number
ADA238790

Entities

People

  • Benjamin F. Dickens

Organizations

  • George Washington University

Tags

Communities of Interest

  • Biomedical

DTIC Thesaurus Topics

  • Alcohols
  • Alkenes
  • Carbon Tetrachloride
  • Cells
  • Chemical Synthesis
  • Chemistry
  • Chlorinated Hydrocarbons
  • Endothelial Cells
  • Free Radicals
  • Halogenated Hydrocarbons
  • Hydrocarbons
  • Membrane Lipids
  • Microsomes
  • Muscle Cells
  • Organic Chemistry
  • Smooth Muscle

Readers

  • Analytical Chemistry
  • Immunology and Pathology
  • Toxicology/Environmental Toxicology