Variations in Thymocyte Susceptibility to Clonal Deletion During Ontogeny: Implications for Neonatal Tolerance
Abstract
For mature T cells, the predominant outcomes of TCR occupancy include secretion of lymphokines and entry into the growth cycle. For many thymocytes, however, receptor occupancy leads to clonal inactivation. This process, known as negative selection, is fundamental to the establishment of immune self- tolerance. Two types of inactivation have been observed, clonal anergy and clonal deletion. Clonal anergy occurs when potentially autoreactive cells persist but fail to respond to self-Ag by secreting lymphokines and proliferating. Clonal deletion is the active elimination of the autoreactive cells and, in several experimental systems using thymocytes, has been shown to be the result of programmed cell death (apoptosis). Some of the hallmarks of activation-induced T cell programmed cell death are a requirement for extracellular Ca2+ and new mRNA and protein synthesis, the autofragmentation of nuclear DNA into 180-to 200-bp multimers, and prevention of the process by cyclosporine A.
Document Details
- Document Type
- Technical Report
- Publication Date
- Jul 15, 1991
- Accession Number
- ADA240997
Entities
People
- Allan M. Weissman
- Carl H. June
- Charles M. Zacharchuk
- Jonathan D. Ashwell
- Mladen Mercep
Organizations
- Naval Medical Research Center